Spreading The Word About TONIGHT TUESDAY APRIL 3rd 7pm ...

Spreading The Word About TONIGHT TUESDAY APRIL 3rd 7pm Meeting about the benefits of total access of LEGAL SHIELD !!

Spread The Word About TONIGHT TUESDAY APRIL 3rd 7pm Meeting about the benefits of total access of LEGAL SHIELD !!

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92% Beauty and the Beast

All Critics (84) | Top Critics (24) | Fresh (84) | Rotten (7) | DVD (36)

The greatest animated film ever made and one of the screen's great musicals hardly needs this (3D) sort of sprucing up.

What you gain in an extra, faked dimension you lose in lively, genuine beauty.

The set pieces are narcotically pleasing, especially the Busby Berkeley-style dancing-kitchenware spectacular, "Be Our Guest," and the romantic ballroom centerpiece that brings Beauty and her Beast together.

The 3-D pops out to enhance the drama or energy of scenes in which settings are large and integral to the action.

Some youthful memories are better not revisited, but this definitely isn't one of them. Sometimes you can go home again.

It is a surprise, in a time of sequels and retreads, that the new film is so fresh and altogether triumphant in its own right.

I admit it: I wrote this more than 20 years ago. But, like " Beauty and the Beast" itself, I think it stands the test of time.

Disney, please understand ... what people are actually showing up for ... they just want to see classic Disney on the big screen. The 3D is incidental.

Disney's gorgeous 1991 animated version of the classic fairy tale "Beauty and the Beast" gets the same 3D treatment that was recently given to "The Lion King."

Watching this in a theater definitely makes it feel less like a "cartoon" and more like a significant film. Cogsworth poking Le Fou in the butt with a sword aside.

One of the brightest jewels in Disney's crown, Beauty and the Beast's 3D reissue takes nothing away and makes the film's brilliance even easier to appreciate; the movie is more beautiful and timeless than it ever was.

Beauty and the Beast is just as enchanting 20 years after its initial release.

The apex of an art form, a justly celebrated classic, and the best animated movie of any sort ever put on screen at any time.

The lines have begun to show in Belle's tale, which remains enjoyable but feels more like a quaint artifact than the masterpiece it once was declared to be.

Both TANGLED EVER AFTER and BEAUTY AND THE BEAST are sure to enthrall audiences everywhere, even if the 3D doesn't really add much of anything to this animated classic.

A serviceable version of a classic fairy tale minus that crucial touch of magic.

And it is a joy to revisit the timeless pleasures of traditional Disney storytelling, with no attempts to add sizzle from celebrity voice talent or radio-friendly pop songs.

3D doesn't downplay the inherent artistry of the effort, but it doesn't enhance anything outside of ticket prices.

Beauty and the Beast looks beautiful and is sure to entertain, but this experience is superfluous at best-it was already a three-dimensional story long before these new technics.

Better remembered than seen, Beauty and the Beast has been treated unkindly not just by the years that have passed since it was released to enormous acclaim in 1991, but by a faddish 3D conversion.

More Critic Reviews

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Galaxy merger to create lithium giant

The pending Galaxy Resources' merger with Canadian explorer Lithium One will create the largest pure play lithium producer by market value in the world, according to the Australian company's managing director Iggy Tan.

Under Galaxy's March 15 offer each Lithium One share would be traded for 1.8 fully paid ordinary Galaxy shares.

Lithium One has a cash balance of about $C10million and controls the lithium potash brine Sal de Vida development, which sits adjacent to one of the largest producers in the world, in Argentina.

Advertisement: Story continues below

Galaxy, which controls the Mt Cattlin mine in Western Australia, a plant in eastern China's Jiangsu province and an adjacent Lithium-Ion Battery Project, has a market capitalisation of about $320 million and is in the process of raising $50 million.

The miner started book-building on Friday, with an announcement expected this week on the outcome.

"What we are doing with the merger with Lithium One and potential capital raising of a minimum of $50 million we are looking at building a pure play lithium company with close to half a billion dollar market cap," Mr Tan said.

"We will be the largest market cap pure lithium company."

The merged company would also become the largest producer of battery-grade lithium, and the fourth or fifth largest producer in the broader lithium industry.

Lithium's role

Lithium mines typically take about five years to come online, but demand is surging for the commodity, Mr Tan said.

"Our view is that you need to find those resources, you need to bring them into production, in anticipation of this demand that's coming," he said.

"The lithium battery is now in your laptops, your mobile phones, your iPads. It's now moving into power tools.

"You can see that growth already has given about a 25 per cent year-on-year growth and think of how many iPads are sold today,? he said, referring to the popular Apple tablet computer.

The world uses about 140,000 tonnes of lithium carbonate a year, which Mr Tan believes will triple in the next decade.

Galaxy shareholders are expected to vote on the deal May 17.

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Researchers uncover new clues to the development of blood and other cancers

Public release date: 2-Apr-2012
[ | E-mail | Share ]

Contact: Diana Quattrone
Diana.Quattrone@fccc.edu
215-815-7828
Fox Chase Cancer Center

Latest findings from Fox Chase Cancer Center reveal that targeting inflammatory pathways may lead to new therapies

CHICAGO, IL (April 2, 2012)Scientists at Fox Chase Cancer Center have uncovered more details about how defects in components of the machinery that makes new proteins can lead to blood and other cancers. The findings, which will be presented at the AACR Annual Meeting 2012 on Monday, April 2, may one day lead to new targeted therapies that address those problems.

"These findings help explain how mutations in one class of proteins can trigger the development of cancer," says Shuyun Rao, Ph.D., a scientific associate in the lab of David L. Wiest, Ph.D., also a co-author on the study, at Fox Chase Cancer Center in Philadelphia. "If we find a way to block the pathway activated by these mutations, this may cause tumors to regress."

The research focused specifically on ribosomal proteins. Previous research has linked mutations in ribosomal proteins to cancers such as leukemia and lymphoma, as well as myelodysplastic syndromes (MDS), which can lead to acute myelogenous leukemia (AML). However, this study represents the first insights into how mutations in ribosomal proteins might increase cancer risk.

To investigate further how an individual ribosomal protein might trigger cancer, Rao, Wiest and their colleagues focused on one known as L22. To begin, they looked at blood samples from a small number of leukemia patientsapproximately 50 and found that in 9%, L22 was either mutated or deleted entirely. This suggested that problems in L22 may have played a role in the development of their cancers.

Next, they deleted L22 in mice that were bred to be prone to develop lymphomas, and saw that their tumors developed faster, and the mice died faster than those who had intact forms of L22 further evidence of its important role in the disease.

Finally, in a sample of cells in the lab, the researchers inactivated L22 and saw that the cells experienced changes, signaling the early stages of lymphoma. Specifically, cells without L22 showed more activity in a pathway associated with inflammation known as NFkappaB, which other research has linked to cancer. "In theory, if we could find a way to block this pathway, we could add this to existing therapies to help treat the tumors it triggers," says Rao.

Although L22 is a ribosomal protein, and therefore helps build new proteins, previous research has found that eliminating L22 does not affect the rate with which cells make new proteins suggesting L22 has additional functions, says Rao.

Previous research has found that L22 expression is strikingly reduced in a number of liquid and solid tumors including breast cancer, lung adenocarcinoma and ovarian carcinoma. "These findings don't just have implications for people with leukemia," Wiest says. "It is likely that L22 repression or inactivation will play a role in other cancer types as well"

###

Co-authors on the study include researchers at Fox Chase, Dana-Farber Cancer Institute in Boston, Massachusetts; St. Jude Children's Research Hospital in Memphis, Tennessee; and the University of Colorado Denver School of Medicine in Aurora, Colorado.

Fox Chase Cancer Center is one of the leading cancer research and treatment centers in the United States. Founded in 1904 in Philadelphia as one of the nation's first cancer hospitals, Fox Chase was also among the first institutions to be designated a National Cancer Institute Comprehensive Cancer Center in 1974. Fox Chase researchers have won the highest awards in their fields, including two Nobel Prizes. Fox Chase physicians are also routinely recognized in national rankings, and the Center's nursing program has received the Magnet status for excellence three consecutive times. Today, Fox Chase conducts a broad array of nationally competitive basic, translational, and clinical research, with special programs in cancer prevention, detection, survivorship, and community outreach. For more information, visit Fox Chase's Web site at www.foxchase.org or call 1-888-FOX CHASE or (1-888-369-2427).

---

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?

AAAS and EurekAlert! are not responsible for the accuracy of news releases posted to EurekAlert! by contributing institutions or for the use of any information through the EurekAlert! system.

Public release date: 2-Apr-2012
[ | E-mail | Share ]

Contact: Diana Quattrone
Diana.Quattrone@fccc.edu
215-815-7828
Fox Chase Cancer Center

Latest findings from Fox Chase Cancer Center reveal that targeting inflammatory pathways may lead to new therapies

CHICAGO, IL (April 2, 2012)Scientists at Fox Chase Cancer Center have uncovered more details about how defects in components of the machinery that makes new proteins can lead to blood and other cancers. The findings, which will be presented at the AACR Annual Meeting 2012 on Monday, April 2, may one day lead to new targeted therapies that address those problems.

"These findings help explain how mutations in one class of proteins can trigger the development of cancer," says Shuyun Rao, Ph.D., a scientific associate in the lab of David L. Wiest, Ph.D., also a co-author on the study, at Fox Chase Cancer Center in Philadelphia. "If we find a way to block the pathway activated by these mutations, this may cause tumors to regress."

The research focused specifically on ribosomal proteins. Previous research has linked mutations in ribosomal proteins to cancers such as leukemia and lymphoma, as well as myelodysplastic syndromes (MDS), which can lead to acute myelogenous leukemia (AML). However, this study represents the first insights into how mutations in ribosomal proteins might increase cancer risk.

To investigate further how an individual ribosomal protein might trigger cancer, Rao, Wiest and their colleagues focused on one known as L22. To begin, they looked at blood samples from a small number of leukemia patientsapproximately 50 and found that in 9%, L22 was either mutated or deleted entirely. This suggested that problems in L22 may have played a role in the development of their cancers.

Next, they deleted L22 in mice that were bred to be prone to develop lymphomas, and saw that their tumors developed faster, and the mice died faster than those who had intact forms of L22 further evidence of its important role in the disease.

Finally, in a sample of cells in the lab, the researchers inactivated L22 and saw that the cells experienced changes, signaling the early stages of lymphoma. Specifically, cells without L22 showed more activity in a pathway associated with inflammation known as NFkappaB, which other research has linked to cancer. "In theory, if we could find a way to block this pathway, we could add this to existing therapies to help treat the tumors it triggers," says Rao.

Although L22 is a ribosomal protein, and therefore helps build new proteins, previous research has found that eliminating L22 does not affect the rate with which cells make new proteins suggesting L22 has additional functions, says Rao.

Previous research has found that L22 expression is strikingly reduced in a number of liquid and solid tumors including breast cancer, lung adenocarcinoma and ovarian carcinoma. "These findings don't just have implications for people with leukemia," Wiest says. "It is likely that L22 repression or inactivation will play a role in other cancer types as well"

###

Co-authors on the study include researchers at Fox Chase, Dana-Farber Cancer Institute in Boston, Massachusetts; St. Jude Children's Research Hospital in Memphis, Tennessee; and the University of Colorado Denver School of Medicine in Aurora, Colorado.

Fox Chase Cancer Center is one of the leading cancer research and treatment centers in the United States. Founded in 1904 in Philadelphia as one of the nation's first cancer hospitals, Fox Chase was also among the first institutions to be designated a National Cancer Institute Comprehensive Cancer Center in 1974. Fox Chase researchers have won the highest awards in their fields, including two Nobel Prizes. Fox Chase physicians are also routinely recognized in national rankings, and the Center's nursing program has received the Magnet status for excellence three consecutive times. Today, Fox Chase conducts a broad array of nationally competitive basic, translational, and clinical research, with special programs in cancer prevention, detection, survivorship, and community outreach. For more information, visit Fox Chase's Web site at www.foxchase.org or call 1-888-FOX CHASE or (1-888-369-2427).

---

[ | E-mail | Share ]

?

AAAS and EurekAlert! are not responsible for the accuracy of news releases posted to EurekAlert! by contributing institutions or for the use of any information through the EurekAlert! system.

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Peninsula Energy's ongoing drilling success delivers upgraded uranium resource at Lance

Monday, April 02, 2012 by John Phillips

Peninsula Energy (ASX: PEN) remains on the road to uranium production from the Lance Projects in Wyoming in 2013, (subject to regulatory approval), where the resource continues to grow.

The string of high grade results announced during early 2012 has provided a 24.3% increase in the total JORC Resource to 51.5 million pounds of uranium.

Just as important is the increase in the confidence categories, with a 30.7% boost in the Measured and Indicated Resource to 14.7 million pounds of uranium.

Also - the vanadium Resource increased to 4.9 million pounds V2O5.

Peninsula has been very clear in mapping the exploration road ahead, with two drilling rigs deployed in the Kendrick area drilling with one dedicated to the along strike exploration and one to intersecting the high-grade nose of the roll fronts.

Gus Simpson, executive chairman, commented on the positive news for the company: ?We are very pleased with the resource upgrade and the ongoing success of the drilling program, particularly the outstanding results at Kendrick.

"The drilling continues to convert areas of mineral potential into JORC resources and to enhance resource status from Inferred to Indicated and Measured. This has added significant value to the Lance projects?.

JORC Resource upgrade - the back story

Recent drilling focused on converting resources from Inferred to Indicated in the proposed Kendrick production unit, which is located to the west of the Ross production unit.

Peninsula said that drilling along the Kendrick roll front system is producing consistent thick high-grade intercepts, which has resulted in its prioritisation due to its resource expansion potential and its proximity to the proposed site of the Lance Central Processing Plant.

The drill density and demonstrated continuity of mineralisation at Kendrick has resulted in a high proportion of Inferred Resources being upgraded to Indicated category.

The resource has been calculated by applying a combined constraint of a grade thickness product (GT) of 0.2 contour and 200ppm U3O8. These cut offs are considered to be appropriate for both calculating and reporting of in-situ recovery (ISR) resources at the Lance Project.

Resource breakdown

Within the Ross Production Unit there is a combined Measured, Indicated and Inferred Resource of 9.0 million pounds uranium with an average grade of 525ppm and an average GT of 0.55.

At Kendrick the combined Measured, Indicated and Inferred Resource totals 29.1 million pounds uranium at an average grade of 494ppm and an average GT of 0.47.

At Barber the combined Measured, Indicated and Inferred Resource totals 13.4 million pounds uranium at an average grade of 446ppm and an average GT of 0.43

Lance projects, development plan

The plan for the Lance projects To build a 2.18mlbs per year ISR operation inclusive of:

? Ion exchange facility, centralised resin stripping ,drying and packaging plant at Ross (CPP).
? Remote ion exchange facility at Barber trucking resin to CPP.

Production plan

Commence production in 2012/13:

? Phase 1 - 750klbs p.a. Ross production unit;? Capex. $63 million.
? Phase 2 ? 750klbs p.a. Kendrick production unit;? Capex. $21 million.
? Phase 3 ? 750klbs p.a. Barber production unit;? Capex. $60 million.
? Production expansion target of 3mlbs per year by 2017.

?

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Islamist militants kill 10 Yemeni soldiers

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Medicare Supplemental Insurance-Health is Now Not as Costly ...

Posted on 31 March 2012. Tags: exit link, health care expenses, hectic days, massive heart attack, medicare supplement plan, medicare supplemental insurance, private health insurance

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